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Journal of Case Reports
Coexistence of Central and Nephrogenic Diabetes Insipidus in a Preterm Infant

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Michel-Macías Carolina1, Cordero-González Guadalupe2
1Neonatology Fellow, Instituto Nacional de Perinatología, México; 2Neonatal Intensive Care Unit, Instituto Nacional de Perinatología, México.
Corresponding Author:
Dr. Michel-Macías Carolina
Email: dra.carolinamichel@gmail.com 
Received: 09-OCT-2016 Accepted: 24-JAN-2017 Published Online: 25-FEB-2017
DOI: http://dx.doi.org/10.17659/01.2017.0022
Background: Diabetes insipidus (DI) is a rare cause of hypernatremia in preterm newborns. Common causes of central DI include intra-ventricular hemorrhage, congenital infection and midline defects. Nephrogenic DI (NDI) is less frequent and can be caused by nephrocalcinosis. Case Report: We hereby report 665 gram female infant born at 27 weeks gestation with central DI and treated with desmopressin. The infant presented again with hypernatremia and showed a poor response to desmopressin. The finding of nephrocalcinosis oriented us to a nephrogenic component of DI. Conclusion: Although nephrogenic diabetes insipidus is the less frequent form of DI, preterm newborns present many risk factors for developing nephrocalcinosis (prolonged ventilation, diuretic therapy, dexamethasone, hypercalcemia) and NDI as a result. In a preterm newborn with CDI, a nephrogenic component must be suspected in the set of decreasing response to DDAVP.
Keywords : Diabetes Insipidus, Diuretics, Hypernatremia, Infant, Nephrocalcinosis.
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